Priming apoptosis
نویسندگان
چکیده
منابع مشابه
Primed to perish: heightened mitochondrial priming explains hESC apoptosis sensitivity.
Human embryonic stem cells (hESCs) are hypersensitive to apoptotic stimuli, though the underlying mechanisms are poorly characterized. In this issue of Cell Stem Cell, Liu et al. (2013) report that mitochondria of human ESCs exist in an apoptosis-prone state, ready to act as cellular executioners upon detecting DNA damage.
متن کاملPriming of immature thymocytes to CD3-mediated apoptosis by infection with murine cytomegalovirus.
Cytomegalovirus (CMV) causes severe clinical manifestations in immunocompromised hosts; however, it remains unclear whether the virus itself is a cause of immunosuppression or whether it is involved as an opportunistic bystander pathogen. This study was performed to elucidate the effect of CMV infection on the host's immune system. The double-positive thymocytes of BALB/c mice inoculated with a...
متن کاملAntigen-Specific Priming is Dispensable in Depletion of Apoptosis-Sensitive T Cells for GvHD Prophylaxis
Prophylactic approaches to graft versus host disease (GvHD) have employed both phenotypic reduction of T cells and selective elimination of host-primed donor T cells in vitro and in vivo. An additional approach to GvHD prophylaxis by functional depletion of apoptosis-sensitive donor T cells without host-specific sensitization ex vivo showed remarkable reduction in GHD incidence and severity. We...
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Twenty-six European Conference on Visual Perception
متن کاملVisuomotor Priming
Two experiments were performed to explore a possible visuomotor priming effect. The participants were instructed to fixate a cross on a computer screen and to respond, when the cross changed colour (“go” signal), by grasping one of two objects with their right hand. The participants knew in advance the nature of the to-be-grasped object and the appropriate motor response. Before (100 msec), sim...
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ژورنال
عنوان ژورنال: Nature Reviews Clinical Oncology
سال: 2013
ISSN: 1759-4774,1759-4782
DOI: 10.1038/nrclinonc.2012.241